Stopping Harmful Oral Bacteria in Their Path The best way to keep bacteria from doing any damage is to cease them in their tracks before they can start down their pathological road to destruction. It was discovered that an adhesin protein molecule, called FadA, in the genes of F. nucleatum. This adhesin, or binding agent, on the bacteria lets them connect with receptors on epithelial cells in the mouth & later the endothelial cells of the placenta. In some way, the receptors on the host cell activate a signal that puts in to action a cascade of processes that permit the bacteria to penetrate the epithelial & endothelial linings & then colonize. According to multiple research in to the mechanisms of bacterial transport not only has potential to prevent preterm & stillborn births, it may have implications in stopping periodontal illness. Periodontal illness has been linked to such health issue as arthritis, diabetes & heart illness. One time it leaves the mouth, the invasion of the bacteria through the placenta allows the bacteria to multiple quickly in the immune-free surroundings that protects the fetus from being rejected by the mother's body. The quick bacterial growth causes the placenta to become inflamed. In turn, the inflammation can trigger preterm birth & fetal death. |
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